[25/03 6:14 PM] Dr Surendra A. Soni:  Is it classical Prameha pidaka   ? Some associated or supurative skin lesions...? Case of DM. He had uncontrolled BS 400+. Now after routine OPD management more 2 months BS is 250+. [25/03 7:51 PM] Dr Ranga Prasad Bhat:+919841218802  Pitakas as of my understanding will be vesiculo-pustular in nature and broad at its base with a narrow mukha. Plz correct me if I'm wrong.  [25/03 8:06 PM] Dr. D. C. Katoch: +919968076668  Dr Soni, the case appears to be of Lichen planus if itching is there, Ayurvedically Alasak. Such a condition irrespective of biochemistry results due to autoimmune disturbance with psychological background. [25/03 8:10 PM] Dr. D. C. Katoch sir: Manjishthadi kwath orally and topical application of Neem taila or Dermin oil of Dr Vasishth work successfully. Add Tagra or Jatamansi or Mental Relaxation technique, if stress factor is apparently present. [25/03 8:17 PM] Dr Surendra A. Soni: Thanks sir..

Consideration of various neurological terminologies are always confusing for the students of Ayurveda, hence i have tried to collect some classical references along with neurological pathogenesis. These are very much similar or close in meaning and basic pathology..... 1.Athetosis-                उद्वेपन (च. सू. 25/40)                वेपनम् ! वेपनमेकदेशे व्यक्तकम्पनम् ।                 Chakrapani on ch chi 24/102 2. Chorea-              अंगविक्षेपणं नर्तन in vatik unmad 3. Dystonia-                       उद्वेष्टन ch su 20/11                       परिवेष्टन ch chi  28/167                        गात्रवेष्टन ch si 7/49 4. Tics-           स्फुरण 5. Tremor-       कम्प 6. Focal dystonia-  शिरो-नासा- अक्षि-जत्रूणां-ग्रीवायाश्च हुण्डनं ।                            ch chi 28/21 6 -A.  Blepharospasm dystonia- वर्त्म स्तंभ/संकोच ch su         20/11 निमेषः चालयत्यति वर्त्मानि su u 3/25 6-B. Oromandibular dystonia-

[20/03 7:43 AM] Prof. Satyendra ojha sir: CHOLANGIOCARCINOMA (CCC) CCC typically refers to mucin-producing adenocarcinomas that arise from the bile ducts. They are grouped by their anatomic site of origin as intrahepatic, hilar (central, ~65% of CCCs), and peripheral (or distal, ~30% of CCCs). They arise on the basis of cirrhosis, excepting primary biliary cirrhosis. Aetiology Although most CCCs have no obvious cause, several predisposing factors have been identified, including primary sclerosing cholangitis, an autoimmune disease (10–20% of PSC patients), and liver fluke in Asians, especially Opisthorchis viverrini and Clonorchis sinensis. CCC seems also to be associated with any cause of chronic biliary inflammation and injury, with alcoholic liver disease, choledocholithiasis, choledochal cysts (10%), and Caroli’s disease. Clinical Features: CCC most typically presents as painless jaundice, often with pruritus or weight loss, and acholic stools. Investigation: