Article-series: AYURVEDIC MANAGEMENT OF PITTODARA ASSOCIATED WITH JIRNA ATISARA (ULCERATIVE COLITIS): A SINGLE CASE STUDY
AYURVEDIC MANAGEMENT OF PITTODARA ASSOCIATED WITH JIRNA ATISARA (ULCERATIVE COLITIS): A SINGLE CASE STUDY
Vaishali Lalwani¹, Shubham
Tripathi², Soni Surendra³, Soni Anamika⁴
- 2nd
Year MD Scholar, An Upgraded PG Department of Kayachikitsa, Govt. Akhandanand
Ayurved College, Ahmedabad, Gujarat
- 2nd
Year MD Scholar, An Upgraded PG
Department of Kayachikitsa, Govt. Akhandanand Ayurved College, Ahmedabad,
Gujarat
- Professor
and Head, An Upgraded PG Department of Kayachikitsa, Govt. Akhandanand
Ayurved College, Ahmedabad, Gujarat
- Associate
Professor, An Upgraded PG Department of Kayachikitsa, Govt. Akhandanand
Ayurved College, Ahmedabad, Gujarat
ABSTRACT
Ulcerative Colitis (UC) is a chronic
inflammatory bowel disease characterized by continuous mucosal inflammation,
ulceration and rectal bleeding. From an Ayurvedic perspective, the
present case was correlated with Pittodara, a form of Udara Roga
associated with aggravated Pitta, Agni Dushti, Ama formation,
Srotodushti and Dhatu Kshaya. A 42-year-old female presented with
20–25 episodes/day of mucoid watery stools with occasional bleeding, abdominal
heaviness, distension, weakness and progressive weight loss. Colonoscopy showed
ulcerative pancolitis with continuous inflammation throughout the colon, and
biopsy confirmed chronic moderately active ulcerative colitis. The patient
received Shamana Chikitsa with Kutaj Ghanavati, Sanshamani Vati,
Sudarshan Ghanavati and Dadimadi Ghrita, along with Tikta Skandha
Kshira Basti for 40 days. The treatment was planned on principles of Deepana,
Ama Pachana, Stambhana, Srotoshodhana, Vrana Ropana and Brimhana. Bowel
frequency decreased to 2–3 episodes/day, with complete resolution of mucous
discharge and rectal bleeding, improvement in appetite and strength, and weight
gain. Follow-up colonoscopy demonstrated marked mucosal healing and
near-complete endoscopic remission, with Mayo score improving from 3 to 1 and
UCEIS from 6–8 to 2. No treatment-related adverse events were reported. This
single case suggests that Samprapti
Vighatana-based Ayurvedic approach may have potential as a complementary
management strategy in chronic active ulcerative pancolitis; however, larger
controlled studies are required.
KEYWORDS: Pittodara; Ulcerative Colitis;
Kshira Basti;Samprapti Vighatana; Ayurveda.
INTRODUCTION
Ulcerative Colitis (UC) is a chronic inflammatory
bowel disease characterized by continuous mucosal inflammation that invariably
begins in the rectum and extends proximally in a continuous pattern,
potentially involving the entire colon (ulcerative pancolitis).
Histopathologically, the inflammation is confined to the mucosa and lamina
propria and is characterized by cryptitis, crypt abscess formation, goblet cell
depletion, and superficial ulceration. Clinically, patients commonly present
with recurrent episodes of bloody diarrhoea, rectal bleeding, mucopurulent
stools, abdominal pain, and urgency, with disease severity varying according to
the extent of colonic involvement. Chronic inflammation may subsequently result
in pseudopolyps, colonic shortening, fibrosis, and dysplastic changes,
emphasizing the importance of timely and effective therapeutic intervention.
[1]
From an Ayurvedic perspective, chronic
gastrointestinal disorders such as Atisara, Pravahika, Grahani,
and Arsha, when neglected or inadequately managed, ultimately progress
towards the spectrum of Udara Roga, particularly in the presence of Dhatu
Kshaya and persistent Agnimandya. The present case demonstrates
features consistent with Pittodara, a subtype of Udara Roga, in
which aggravated Pitta Dosha, associated with Vata and Kapha,
produces Srotorodha and significantly impairs Jatharagni at the
level of the Aamashaya and Mahasrotas. [3]
The underlying pathology may be understood as a
chronic persistence of Amla Avastha Paka, resulting in sustained
derangement of digestive and metabolic processes. Progressive Agnidushti
leads to Ama formation, Pitta Prakopa, Rakta Dushti, and
mucosal injury, producing ulceration, bleeding, and inflammatory changes within
the colon. With increasing chronicity, the disease extends beyond the Koshtha
(gastrointestinal tract) to involve the Shakha (peripheral tissues), Marma-Asthi-Sandhi
(vital organs and structural tissues), and Satva (mental well-being),
transforming a localized gastrointestinal disorder into a systemic disease
process. [3]
Considering these pathological principles, Ayurvedic
management aims not only at symptomatic relief but also at restoring Agni,
eliminating Ama, correcting Dosha imbalance, promoting Srotoshodhana,
facilitating Vrana Ropana (mucosal healing), and replenishing depleted
tissues (Dhatu Poshana), thereby addressing the disease at its root and
reducing the likelihood of recurrence. [3]
CASE PRESENTATION
A 42-year-old female presented with a history of
chronic gastrointestinal complaints with progressive worsening of symptoms. The
chief complaints included:
- Picchila
Drava Mala Pravritti (frequent passage of mucoid
watery loose stools; 20–25 episodes/day) since 10-15 years
- Udara
Gauravata & adhman(persistent
abdominal heaviness) since 4-5 years
- Daurbalyata
(generalized weakness and easy fatigability) since 4-5 years
- Sharira
Bhara Hani (progressive weight loss) since
4-5 years
- Jaliya
Nasa Srava (watery nasal discharge) since 1
month
The patient had significant impairment in daily
activities due to increased bowel frequency and associated constitutional
symptoms.
Investigations
Colonoscopy -29/08/25
- Diffuse
ulcerative pancolitis with continuous mucosal inflammation involving the
entire colon, extending from the rectum to the proximal colon.
Histopathological Examination
- Colonic
biopsy revealed features consistent with chronic moderately active
ulcerative colitis, confirming the diagnosis.
Personal History
- Ahara
Shakti (Appetite): Reduced
- Mala
Pravritti (Bowel Habits): Frequent mucoid
watery loose stools (20–25 episodes/day)
- Mutra
Pravritti (Micturition): Normal
- Nidra
(Sleep): Disturbed
- Vyasana
(Addiction): None
- Ahara
Vihara: Irregular dietary habits with
poor nutritional intake secondary to chronic illness.
SAMPRAPTI (PATHOGENESIS)
Nidana
Sevana
(Pitta-provoking diet, Vidahi Ahara, incompatible food, and faulty lifestyle)
⬇
Pitta Prakopa with Vata–Kapha Anubandha
(Aggravation of Pitta associated with Vata and Kapha)
⬇
Srotorodha & Unmarga Gamana
(Obstruction of microchannels and derangement of the normal physiological
pathways)
⬇
Jatharagni Dushti in Aamashaya
(Impairment of digestive fire leading to Agnimandya)
⬇
Disturbance of Mahasrotas Avastha Paka
(Persistent derangement of digestion with chronic Amla Avastha Paka and
Ama formation)
⬇
Formation of Sama Pitta & Sama Rasa &Rakta Dhatu
(Generation of inflammatory toxic metabolites with vitiation of Rasa &Rakta
Dhatu)
⬇
Vyapti to Shakha, Marma, Asthi-Sandhi & Satva
(Systemic dissemination of pathology beyond the gastrointestinal tract)
⬇
Dhatu Poshana Avarodha & Progressive Dhatu Kshaya
(Impaired tissue nourishment resulting in progressive tissue depletion)
⬇
Karshya, Daurbalya & Ojakshaya
(Weight loss, generalized weakness, and reduced vitality)
⬇
Sthanika Dushya-Dushti in Purishavaha Srotas
(Localization of pathology in the colonic mucosa)
⬇
Shotha • Daha • Rakta Srava • Picchila Mala Pravritti • Adhmana
(Mucosal inflammation, burning sensation, rectal bleeding, mucoid diarrhea,
and abdominal distension)
⬇
PITTODARA
According to Ayurveda, the pathogenesis of Pittodara
is a dynamic and progressive process that originates from the persistent
indulgence in Pitta-provoking Nidanas. Excessive consumption of Katu
(pungent), Amla (sour), Lavana (salty), Ushna (hot), Tikshna
(penetrating), and Vidahi (irritant) food substances, along with
incompatible dietary practices (Viruddha Ahara), Adhyashana
(consuming food before the previous meal is digested), irregular eating habits,
prolonged exposure to heat or sunlight, psychological stress, and pre-existing
gastrointestinal disorders collectively initiate the pathological process.
Individuals possessing Pitta Prakriti, Rakta Sara, younger age,
or a predisposition to gastrointestinal disorders are particularly susceptible
to the development of this condition. [3]
Acharya Charaka
has explained the initial stage of this pathological cascade through the fundamental
principle of "Raso Nipate Dravyanam," wherein ingested
substances exert their direct influence upon the gastrointestinal mucosa
immediately after contact. Under physiological conditions, the protective
action of Kledaka Kapha and the mucosal barrier safeguards the
gastrointestinal lining from chemical injury. However, persistent exposure to Pitta-provoking
substances overwhelms these protective mechanisms, resulting in progressive
mucosal irritation, congestion, inflammation, epithelial injury, and
ulceration. This stage bears a remarkable resemblance to the early pathological
events of ulcerative colitis, where disruption of the epithelial barrier
initiates continuous mucosal inflammation. [4]
As the disease progresses, aggravated Pitta Dosha
associates with Vata (Anila) and Kapha, resulting in a
complex Tridoshaja pathological state with predominant Pitta. The
synergistic vitiation of these Doshas leads to Srotorodha
(obstruction of physiological channels) and Unmarga Gamana (deviation
from normal physiological pathways). Consequently, the vitiated Doshas
localize in the Aamashaya, where they impair Jatharagni, that may
manifest as Tikshnagni, Vishamagni, and eventually Mandagni.
Failure of Agni disrupts the normal sequential process of Avastha
Paka, causing persistent retention and predominance of Amla Avastha Paka
within the Mahasrotas. [3]
The chronic derangement of Avastha Paka
initiates the formation of Ama, Sama Pitta, and Sama Rasa&
Rakta Dhatu. This toxic metabolic complex establishes a sustained
inflammatory milieu within the gastrointestinal tract. From a contemporary
perspective, this stage may be correlated with persistent oxidative stress,
intestinal dysbiosis, disruption of epithelial tight junctions, activation of
inflammatory cytokines, excessive immune responses, and continuous mucosal
injury. Thus, the pathological process gradually transforms from a functional
disturbance of digestion into an established chronic inflammatory disease. [3]
Persistent Agnimandya together with repeated
episodes of diarrhoea severely compromises Dhatu Poshana. The continuous
loss of nutrients, impaired digestion, and defective tissue metabolism
progressively arrest the normal sequence of nourishment from Rasa to
subsequent Dhatus. Consequently, Rasa, Rakta, Mamsa,
and Meda Dhatus undergo progressive Kshaya, producing clinical
manifestations such as Karshya (weight loss), Daurbalya
(generalized weakness), anorexia, fatigue, and reduced vitality.
Simultaneously, depletion of Kapha weakens the protective integrity of
the intestinal mucosa, whereas accumulated Vata further aggravates
intestinal motility and structural instability, perpetuating the disease
process. [3]
Because of the chronicity of the disease, the
pathological process is no longer confined to the Koshtha. The
circulating Sama Pitta and Sama Rasa Dhatu disseminate through
the Mahasrotas into the Shakha, Marma, Asthi-Sandhi,
and Satva. This transition represents the evolution of a localized
gastrointestinal disorder into a systemic pathological process involving
peripheral tissues, vital structures, and the psychoneuro-immunological axis.
The involvement of Satva explains the close relationship between
emotional stress and disease exacerbation, which is well recognized in patients
with ulcerative colitis. [3]
Ultimately, the pathological process undergoes Sthana
Samshraya within the Purishavaha Srotas, where severe Sthanika
Dhatu Dushti develops. The vitiated Pitta predominantly affects Rakta
Dhatu, producing vascular congestion, increased capillary fragility, and
recurrent rectal bleeding (Rakta Srava). Simultaneously, Mamsa Dhatu
involvement results in mucosal erosion and ulcer formation (Vrana),
while Kapha Dushti contributes to excessive mucous secretion (Picchila
Srava). Vata aggravation further manifests as abdominal pain,
urgency, tenesmus, and altered bowel motility. The combined effects of Shotha
(mucosal inflammation), Daha (burning sensation), Rakta Srava, Picchila
Drava Mala Pravritti, and Adhmana closely resemble the pathological
and clinical features of chronic active ulcerative pancolitis, including
diffuse mucosal inflammation, cryptitis, crypt abscess formation, goblet cell
depletion, and continuous ulceration of the colonic mucosa. [3]
From an Ayurvedic standpoint, frequent diarrhoeal
episodes observed in Pittodara may be interpreted as the body's
intrinsic attempt to eliminate accumulated Ama and aggravated Pitta
Dosha from the Mahasrotas. [3]
Pittodara
Symptom - เคคเคธ्เคฏ เคฐूเคชाเคฃि- เคฆाเคนเค्เคตเคฐเคคृเคท्เคฃाเคฎूเคฐ्เค्เคाเคคीเคธाเคฐเคญ्เคฐเคฎाः [Cha.Chi.13/22] [3]
This slok represents a chronological pathological
cascade, tracking the disease from localized gut inflammation to systemic
neurological collapse. [3]
The Samprapti of Pittodara can be
summarized as:
Pitta Prakopaka Nidana Sevana
→ Pitta Prakopa with Agni Dushti → Chronic dominant Amla
Avastha Paka → Formation of Sama Pitta and Sama Rasa Dhatu → Srotodushti
and impaired Dhatu Poshana → Progressive Dhatu Kshaya → Picchila
Drava Mala Pravritti, Udara Gauravata, Daurbalya, Karshya
→ Pittodara (Ulcerative Pancolitis). [3]
This Samprapti demonstrates that chronic
aggravation of Pitta and persistent Amla Avastha Paka initiate a
cascade of Agni Dushti, Ama formation, Srotodushti, and
progressive Dhatu Kshaya, ultimately resulting in severe mucosal injury
of the Purishavaha Srotas. Therefore, successful management depends on Samprapti
Vighatana through Deepana, Ama Pachana, Pitta Shamana,
Stambhana, Vrana Ropana, and Brimhana, thereby restoring Agni,
promoting mucosal healing, and achieving sustained clinical remission. [3]
Before:
29/08/25
Based on:
- Clinical
presentation
- Colonoscopy
Findings
- Ayurvedic
Lakshanas
Diagnosed as Pittodara(correlated with
Ulcerative Colitis)
THERAPEUTIC INTERVENTION
1. Shamana Chikitsa
- Kutaj
Ghanvati Dose 2/2/2/2
- Sansamani
Vati 4/4/4/4
- Sudarshan
Ghanavati 2/2/2/2
- Dadimadi
Ghrita 20 ml empty stomach early morning
2. Panchakarma
Kshira Basti
was selected as the principal Panchakarma intervention owing to its
ability to interrupt the Samprapti of Pittodara at multiple
levels. Since Vata Dosha, particularly Apana Vayu, plays a pivotal
role in the progression and chronicity of Udara Roga, Basti is
regarded as the most effective therapeutic modality for restoring its
physiological function. [7]
Composition of Kshira Basti
- Tikta
Skandha Kwatha – 100 gms
- Kshira
– 200 ml
- Jala
– 800 ml (reduced to 200 ml during preparation) method: ksheerpak
- Madhu
– 20 ml
- Ghrita
– 20 ml
- Temp.
– Room Temp.
Total Volume: 240 ml
The treatment protocol was structured with targeted Shaman
Chikitsa administered continuously for 2 months, supplemented by Tikta
skandha kshir basti for 40 days. [7]
Mechanism of Action
- Regulation
of Apana Vayu: As the foremost therapy for Vata, Tikta
Skandha Kshira Basti restores the normal function of Apana
Vayu, thereby reducing bowel frequency, urgency, and abnormal
intestinal motility. [6,7]
- Direct
Action on the Pakvashaya: Administered through the rectal route, Basti
prepared with pittashamak drugs directly reaches the Pakvashaya—the
principal seat of Vata and the site of pathology in Pittodara.
This enables targeted Samprapti Vighatana at the disease site with
‘raso-nipate dravyanam’ principle. [5,6]
- Removal
of Srotorodha: The Tikta Skandha drugs possess Pitta-Shamaka,
Rakta Prasadana, Shothahara, and Vrana Ropana
properties, facilitating Srotoshodhana, reducing inflammation, and
promoting healing of the ulcerated colonic mucosa. [3,7]
- Pacification
of Pitta and Vata: The combination of Tikta Dravyas
with Kshira and Ghrita provides a balanced therapeutic
effect. While Tikta drugs alleviate Pitta and inflammatory
pathology, Kshira and Ghrita nourish tissues, pacify Vata,
and prevent excessive dryness. [3,7]
- Promotion
of Vrana Ropana and Dhatu Poshana: Kshira and Ghrita
exert mucoprotective [ Brimhana, Jeevaniya, and Rasayana
] effects, supporting regeneration of the damaged intestinal mucosa,
improving tissue nutrition, and correcting Dhatu Kshaya. [3]
- Restoration
of Gastrointestinal Homeostasis: By simultaneously correcting Agni,
reducing Ama, pacifying Doshas, and restoring Purishavaha
Srotas integrity, Kshira Basti promotes sustained mucosal
healing and minimizes disease recurrence. [3,5,6]
Thus, Kshira Basti acts not merely as a
symptomatic therapy but as a comprehensive Samprapti Vighataka
intervention, addressing the fundamental pathological mechanisms of Pittodara
and facilitating long-term clinical remission. [5,6,7]
Assessment of Disease Improvement:
TABLE
2. SUBJECTIVE PARAMETERS BEFORE TREATMENT
|
Symptoms |
Before treatment |
|
Picchila Drava Mala Pravritti |
++++ |
|
Udara Gauravata & adhman |
++++ |
|
Daurbalyata |
+++ |
|
Sharira Bhara Hani |
+++ |
|
Jaliya Nasa Srava |
++ |
TABLE
3. WEEK-WISE IMPROVEMENT IN SUBJECTIVE PARAMETERS
|
Symptoms |
After
treatment |
|||
|
1st
week |
2nd
week |
3rd
week |
4th
week |
|
|
Picchila
Drava Mala Pravritti |
+++ |
++ |
+ |
- |
|
Udara
Gauravata
& adhman |
+++ |
+ |
+ |
- |
|
Daurbalyata |
+++ |
++ |
+ |
- |
|
Sharira
Bhara Hani |
+++ |
++ |
+ |
- |
|
Jaliya
Nasa Srava |
++ |
+ |
- |
- |
Results:
Following the completion of the Ayurvedic
treatment protocol for 40 days , patient demonstrated marked clinical,
nutritional, and endoscopic improvement, indicating successful Samprapti
Vighatana and sustained disease remission.
Clinical Outcomes
- Significant
Reduction in Bowel Frequency: The frequency of bowel movements decreased
from 20–25 episodes/day to 2–3 times/day, well-controlled bowel
movements/day, with complete resolution of urgency and improved bowel
control.
- Restoration
of Normal Stool Characteristics: Stool consistency improved from frequent
mucoid watery diarrhoea to well-formed stools. Both rectal bleeding (Rakta
Srava) and mucous discharge (Picchila Mala) resolved
completely, indicating effective mucosal healing.
- Improvement
in Agni and Appetite: Restoration of Jatharagni was
reflected by a marked improvement in appetite and digestion, with complete
relief from anorexia and improved tolerance to a normal diet.
- Reversal
of Dhatu Kshaya: Progressive weight gain and improvement in
physical strength were observed, indicating restoration of Dhatu
Poshana and correction of chronic tissue depletion.
- Resolution
of Associated Symptoms: Systemic manifestations, including generalized
weakness (Daurbalya), abdominal heaviness (Udara Gauravata),
and recurrent watery nasal discharge (Jaliya Nasa Srava), resolved
completely, resulting in a substantial improvement in the patient's
overall quality of life.
Endoscopic Outcomes
Follow-up colonoscopic evaluation demonstrated
remarkable mucosal healing and near-complete endoscopic remission. The
extensive inflammatory changes involving the entire colon observed before
treatment showed significant regression. The disease improved from severe
ulcerative pancolitis (Montreal Classification E3; Endoscopic Mayo Score 3;
UCEIS 6–8) with diffuse ulceration, spontaneous bleeding, and extensive
mucosal friability to mild residual disease (Endoscopic Mayo Score 1; UCEIS
2). The sigmoid, descending, and transverse colon appeared completely
normal, while only minimal superficial, non-friable erosions persisted in the
rectum and ascending colon, without evidence of active bleeding or significant
inflammatory activity. [1,2]
Overall, the patient achieved clinical remission,
nutritional recovery, and substantial endoscopic remission without any
treatment-related adverse events or complications, highlighting the
effectiveness of the Samprapti-based Ayurvedic management approach in
chronic active ulcerative pancolitis.
A. Objective
Criteria :
Colonoscopy (After
treatment):- Date: 13.10.2025
Discussion:
The favorable clinical and endoscopic outcomes
observed in this case demonstrate the effectiveness of a Samprapti Vighatana-based
Ayurvedic approach in the management of Pittodara correlated with
chronic active ulcerative pancolitis. Rather than providing symptomatic relief
alone, the treatment addressed the fundamental pathological events, including Agni
Dushti, Ama formation, Pitta Prakopa, Srotorodha, and
progressive Dhatu Kshaya. Restoration of Jatharagni, elimination
of Ama, regulation of Apana Vayu, and healing of the ulcerated
colonic mucosa collectively resulted in sustained clinical remission and marked
endoscopic improvement. [1,2,3]
The therapeutic strategy was designed to achieve
multiple objectives simultaneously. It pacified aggravated Pitta and Rakta,
thereby reducing mucosal inflammation, burning sensation (Daha), and
rectal bleeding (Rakta Srava). Correction of Apana Vayu through Kshira
Basti normalized bowel motility, significantly reducing bowel frequency and
urgency. Simultaneously, restoration of the protective Shleshma layer
preserved the integrity of the colonic mucosal barrier, preventing further
ulceration and facilitating tissue regeneration. Correction of Agni
through Deepana and Ama Pachana arrested the continuous formation
of Amla Avastha Paka, while Brimhana therapy restored Dhatu
Poshana, leading to reversal of Mamsa-Meda Dhatu Kshaya, progressive
weight gain, and improved physical strength. [3,7]
Each therapeutic intervention contributed uniquely
to the overall clinical outcome. Kutajghan Vati provided potent Grahi,
Deepana, Pachana, and Pitta-Vata Shamana actions,
effectively controlling chronic diarrhea, mucous discharge, and rectal
bleeding. Sudarshan Ghanavati, owing to its Tikta Rasa and Pachana
properties, pacified aggravated Pitta, improved Jatharagni,
facilitated Ama Pachana, and relieved Srotorodha, thereby
reducing abdominal heaviness and distension. Sanshamani Vati acted as an
effective Pitta-Rakta Shamaka and Rasayana, helping to regulate
systemic inflammatory activity, reduce Daha, and improve associated
constitutional symptoms. [3]
Oral administration of Dadimadi Ghrita played
a crucial role in Vrana Ropana by promoting healing of the ulcerated
colonic mucosa. Its Snigdha, Grahi, and Brimhana
properties protected the intestinal mucosal barrier, reduced bowel
hypermotility, and supported restoration of normal gastrointestinal function.
The principal Panchakarma intervention, Tikta Skandha Kshira Basti,
directly targeted the Pakvashaya—the principal seat of Vata and
the primary site of pathology. The combined action of Tikta Dravyas, Kshira,
and Ghrita facilitated Srotoshodhana, pacified both Pitta
and Vata, nourished depleted tissues, accelerated Vrana Ropana,
and interrupted the pathological cascade at its site of origin. [3]
The remarkable improvement in bowel frequency,
complete cessation of rectal bleeding and mucous discharge, restoration of
appetite, weight gain, and near-complete endoscopic mucosal healing
collectively indicate successful reversal of the underlying Samprapti.
These findings suggest that a comprehensive Ayurvedic treatment protocol
integrating Deepana, Ama Pachana, Pitta-Rakta Shamana, Stambhana, Vrana
Ropana, Brimhana, and Kshira Basti can effectively restore gastrointestinal
homeostasis and induce sustained clinical as well as endoscopic remission in
chronic active ulcerative pancolitis. [1,2]
Overall, this case reinforces the principle that
management directed toward correcting the underlying Samprapti, rather
than suppressing isolated symptoms, has the potential to achieve durable
mucosal healing and systemic recovery in Pittodara correlated with
Ulcerative Colitis.
[3]
This case demonstrates the successful management of
chronic active Ulcerative Pancolitis through a Samprapti Vighatana-based
Ayurvedic approach by correlating the disease with the classical concept of Pittodara.
The pathological process was understood as a consequence of persistent Pitta
Prakopa, Agni Dushti, chronic Amla Avastha Paka, formation of
Sama Pitta and Sama Rasa Dhatu, ultimately leading to Srotodushti,
Dhatu Kshaya, and localized injury of the Purishavaha Srotas.
This comprehensive understanding formed the basis of the therapeutic strategy
rather than merely addressing symptomatic manifestations. [1,2,3]
The integrated treatment protocol, comprising Deepana,
Ama Pachana, Pitta-Rakta Shamana, Stambhana, Vrana
Ropana, Brimhana, and Tikta Skandha Kshira Basti, effectively
restored Jatharagni, corrected Apana Vayu, promoted mucosal
healing, and re-established normal Dhatu Poshana. Clinically, this was
reflected by normalization of bowel frequency, complete cessation of rectal
bleeding and mucous discharge, restoration of appetite, progressive weight
gain, and resolution of associated systemic symptoms. Furthermore, follow-up
colonoscopy demonstrated marked mucosal healing with near-complete endoscopic
remission, indicating that the therapeutic response extended beyond symptomatic
relief to objective tissue recovery. [3,7]
Conclusion:
The present case highlights the potential of
classical Ayurvedic principles to address both the local inflammatory pathology
and the underlying systemic metabolic derangements involved in Ulcerative
Colitis. By targeting the root Samprapti, Ayurveda may provide a
rational, safe, and holistic therapeutic approach capable of achieving
sustained clinical and endoscopic remission. [1,2,3]
However, as these observations are based on a single
case, larger prospective clinical studies with standardized treatment
protocols, objective biomarkers, endoscopic assessment, and long-term follow-up
are warranted to further validate the efficacy and reproducibility of this
Ayurvedic management strategy in patients with Ulcerative Colitis.
Conflict of Interest Statement :
None declared.
----------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------
Above article has been published in 'International Journal of Ayurveda & Pharma Research
Article Link-
https://ijapr.in/index.php/ijapr/article/view/4410
Please visit above link for full article.
----------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------
Presented by
*Dr. Vaishali Lalwani
B.A.M.S.
MD IInd Yr. Scholar
P. G. Dept. of Kayachikitsa
Govt. Akhandanand Ayu. College,
Bhadra, Ahmedabad, Gujarat, India
Email: vaishalilalwani93137@gmail.com
**Dr. Shubham Tripathi
B.A.M.S.
MD IInd Yr. Scholar
P. G. Dept. of Kayachikitsa
Govt. Akhandanand Ayu. College,
Bhadra, Ahmedabad, Gujarat, India
Mob.- +91-6260097626
***Guided by
Prof. Surendra A. Soni
H.O.D.,
P. G. Dept. of Kayachikitsa
Govt. Akhandanand Ayu. College,
Bhadra, Ahmedabad, Gujarat, India
Email: kayachikitsagau@gmail.com
Dr. Anamika S. Soni
Associate Professor
P. G. Dept. of Kayachikitsa
Govt. Akhandanand Ayu. College,
Bhadra, Ahmedabad, Gujarat, India
Comments
Post a Comment