Rakta avrita Vata
Vata dosha alongwith ruksha, laghu, shita, khara, suksma guna possesses cala guna.
Yasya prerane shakti sa cala │
Va.Su.1/40 (Arundatta)
Cala guna is essential for avyahatgati of vata dosha and also for the pravartan (stimulation), chesta (movement), vyuhakar (organizing), ksepta (excretion), santan gati (sinus rhythym), pumping and pulsation. Thus whenever cala guna will be affected vikshepa, samvahana, parivahana, chavan, pargaman, sravan, visyandana karma will be hampered.
As understood in grahani adhyaya yugpat, sarvatra, continuous vikshepa of rasa rupa dhatu is by vyanvayu which is responsible for nutrition of sthyayi dhatu. Whenever due to kha vaigunya the vikshepita rasa gets obstructed/ stagnated, pathogenesis takes place. Thus for avyahatgati of vata normalcy of marga and margastha dhatu is essential.
Marga means various channels, srotas, sira, dhamani, rasayani, rasavahini, nadi, pantha, sharirchidra, aashaya, niketa etc. whereas margastha dhatu means drava rupa asthayi raktadi dhatu.
Specific ratio of pancamahabhut maintains the dravatva of dhatu. More the viscosity slow is the flow of the drava rupa dhatu. Viscosity will increase whenever parthivata will increase in proportion in margastha dhatu. Change in specific proportion is primarily due to agni. It may be at level of jatharagni, dhatvagni or bhutagni. Secondly, anupahat agni is responsible for maintaining of normalcy of dhatus. Agnimandya leads to apachit dhatu vridhi. Such apachit dhatu are nothing but aam which may act as antigen. Vyadhi vighatkar bhava comes in action to prevent adherence of aam with specific dhatu.
Thus presence of aam and vyadhi vigatkar bhava changes the specific ratio of dravatva leading to reduction of flow (Saratva) or capillary perfusion and increase in organ congestion and syndromes of hyperviscosity.
Therefore upahata agni causes variation in dravata leading to obstruction or aavaran of vata dosha causing aavrita vata.
Secondly anupahat srotas is necessary for dhatu poshan. Srotas over here means marga or channels. If the patency of channels is hampered it disturbs the flow of drava rupa dhatu. Patency of channels depends on bija (genetic), environmental factors (external factors) and internal environment within the channels.
It is important to understand the significance why Kush Sankrityayana while explaining 6 qualities did not explain cala as quality of vata whereas he explained daruna as quality of Vata. Secondly answer given to question of Kankayan Rishi on how asanghata or amurta vata gets prakopita or prashaman by murta dravya.
Acharya Badish Dharmargava has explained that ruksa, laghu, shita, khara, vishada, sushira and daruna guna acts on sharir and as vayu takes ashraya of sharir for its activity prakopa of vayu takes place whereas snigdha, guru, ushna, slakshna, mridu, picchila guna when increase in sharir they do prashaman of ashrayi vata dosha. Therefore darunata i.e. hardness of marga causes prakopa of vata. Specific hardness of channels is essential for normal flow. If such darunata is lost it causes aneurysm leading to impaired flow of drava rupi dhatu as understood in siragata vata whereas hardness when increased the flow is obstructed causing vataprakopa.
Thus one can conclude from above discussion that for normalcy of vatagati following entities are essential.
1) Specific ratio of drava rupa dhatu ( Viscosity)
2) Marga ( Channels)
Vata dosha alongwith ruksha, laghu, shita, khara, suksma guna possesses cala guna.
Yasya prerane shakti sa cala │
Va.Su.1/40 (Arundatta)
Cala guna is essential for avyahatgati of vata dosha and also for the pravartan (stimulation), chesta (movement), vyuhakar (organizing), ksepta (excretion), santan gati (sinus rhythym), pumping and pulsation. Thus whenever cala guna will be affected vikshepa, samvahana, parivahana, chavan, pargaman, sravan, visyandana karma will be hampered.
As understood in grahani adhyaya yugpat, sarvatra, continuous vikshepa of rasa rupa dhatu is by vyanvayu which is responsible for nutrition of sthyayi dhatu. Whenever due to kha vaigunya the vikshepita rasa gets obstructed/ stagnated, pathogenesis takes place. Thus for avyahatgati of vata normalcy of marga and margastha dhatu is essential.
Marga means various channels, srotas, sira, dhamani, rasayani, rasavahini, nadi, pantha, sharirchidra, aashaya, niketa etc. whereas margastha dhatu means drava rupa asthayi raktadi dhatu.
Specific ratio of pancamahabhut maintains the dravatva of dhatu. More the viscosity slow is the flow of the drava rupa dhatu. Viscosity will increase whenever parthivata will increase in proportion in margastha dhatu. Change in specific proportion is primarily due to agni. It may be at level of jatharagni, dhatvagni or bhutagni. Secondly, anupahat agni is responsible for maintaining of normalcy of dhatus. Agnimandya leads to apachit dhatu vridhi. Such apachit dhatu are nothing but aam which may act as antigen. Vyadhi vighatkar bhava comes in action to prevent adherence of aam with specific dhatu.
Thus presence of aam and vyadhi vigatkar bhava changes the specific ratio of dravatva leading to reduction of flow (Saratva) or capillary perfusion and increase in organ congestion and syndromes of hyperviscosity.
Therefore upahata agni causes variation in dravata leading to obstruction or aavaran of vata dosha causing aavrita vata.
Secondly anupahat srotas is necessary for dhatu poshan. Srotas over here means marga or channels. If the patency of channels is hampered it disturbs the flow of drava rupa dhatu. Patency of channels depends on bija (genetic), environmental factors (external factors) and internal environment within the channels.
It is important to understand the significance why Kush Sankrityayana while explaining 6 qualities did not explain cala as quality of vata whereas he explained daruna as quality of Vata. Secondly answer given to question of Kankayan Rishi on how asanghata or amurta vata gets prakopita or prashaman by murta dravya.
Acharya Badish Dharmargava has explained that ruksa, laghu, shita, khara, vishada, sushira and daruna guna acts on sharir and as vayu takes ashraya of sharir for its activity prakopa of vayu takes place whereas snigdha, guru, ushna, slakshna, mridu, picchila guna when increase in sharir they do prashaman of ashrayi vata dosha. Therefore darunata i.e. hardness of marga causes prakopa of vata. Specific hardness of channels is essential for normal flow. If such darunata is lost it causes aneurysm leading to impaired flow of drava rupi dhatu as understood in siragata vata whereas hardness when increased the flow is obstructed causing vataprakopa.
Thus one can conclude from above discussion that for normalcy of vatagati following entities are essential.
1) Specific ratio of drava rupa dhatu ( Viscosity)
2) Marga ( Channels)
Aahar
v
Agnimandya v
v
Apakva aahar rasa
v
v Dhatusma v (Upahata ushma)
v v
v Sama dhatu [asthayi / dravarupa]
v v
v
Agnimandya v
v
Apakva aahar rasa
v
v Dhatusma v (Upahata ushma)
v v
v Sama dhatu [asthayi / dravarupa]
v v
Vikara vighata bhava v if vighatkara bhava are
v unable to regularize
v (Vyadhyutpatti without--avaran)
v
v
v
v
v
v
Specific ratio of dravata is altered bija dusti and/or environmental factors
v v
v v
v unable to regularize
v (Vyadhyutpatti without--avaran)
v
v
v
v
v
v
Specific ratio of dravata is altered bija dusti and/or environmental factors
v v
v v
^ Viscosity rheological forces Loss of patency of channel
v >>>>>>>>> v
v v
v v
v v
v >>>>>>> Yatra sanga kha vaigunya v (upahat srotas)
v v v
v Vyadhiutpatti without aavaran v
v v
v v
Avarodha to avyahat gati Inflammation of channels
v v
v v
v >>>>>>>>> v
v v
v v
v v
v >>>>>>> Yatra sanga kha vaigunya v (upahat srotas)
v v v
v Vyadhiutpatti without aavaran v
v v
v v
Avarodha to avyahat gati Inflammation of channels
v v
v v
Aavrita vata [upahat srotasgata vayu] v
v v
v v
Vyadhiutpatti without aavaran Vyadhi utpatti with aavara
Rakta Avrita Vata
Raktavrite sa daha arti tvak mansa antarya bhrisam |
Bhavet sa raga swayathu jayante mandalani cha ||
Ca.Ci.28 / 215
In rakta avrita vata there is either quantitative and /or qualitative increase in rakta dhatu which obstructs the gati of vata dosha leading to aavrita vata.
In Vidhishonitiya adhyaya Caraka has explained rakta dustikara hetu which are cause for qualitative and quantitative impairment of rakta dhatu.
Ati lavan rasa sevan causes quantitative increase of rakta
(Raktam Vardhayati)
Kshar causes pachana, daran of the srotas.
Amla rasa does pachana, mansa vidaha
(lepana karma is of mansa and vessels are made up of muscle fibers) and swayathu utpadayati. Rakta dusti, causes inflammation.
Katu rasa reduces bala and has quality to irritate the mucosal lining.
Kulatha has ushna virya and amla vipak. It causes amlapitta and thereby after vitiating pitta causes rakta dusti. Kulatha is mentioned hetu in raktapitta where there is quantitative increase of rakta.
Masha although balya, when taken in excess quantity causes mala vridhi and is ushna in nature.
Tila taila, mulaka, pindalu, jalaja and anup mansa by their ushna guna causes raktadusti.
Sura, souvirak, sukta are ushna and are raktadustikara.
Virudha, upaklinna anna, puti anna, diva swap are agnimandyakar and have low nutritional values.
Aatap and anala sanyog are external factors which directly affect the small blood vessels and are cause for local pathogenesis.
Similarly abhighata/ injury also causes raktadusti as seen in case of superficial venous thrombosis after catheterization.
One thing is common that all the above ahariya dravyas are vatashamaka therefore when taken in excess will hamper gati of vatadosha.
The quantitative increase of rakta causes increase in viscosity and thereby hampering the gati of vata as seen in cases of polycythaemia rubra Vera..
[06/04 7:57 AM] satyendra ojha sir:
Conclusion:
Rakta avritta vata is a process of pathogenesis wherein raktavriddhi (quantitative increase of rakta) impedes the gati of vata which leads to symptoms like pain, redness, burning sensation and localized inflammation.
The symptoms can be observed in various diseases like erythromelalgia, phlebitis, PRV and apparent erythrocytosis. Thus raktavritta vata is not a single disease but initial factor of pathology.
Raktamokshana reduces quantitative increase of blood. Virechan is the best shodhan procedure for raktadusti. Fluid loss due to virechan also has impact on intravascular quantity of fluid plasma does reducing the avarodh.
Shita pradeha mainly medicines like dashanga lepa, kamala, ushira, yastimadhu, sariva, chandan, padmak, darbha etc drugs helps to reduce local inflammatory response. Anti thrombotic effect of darbha, kamala also reduces the avarodh to the gati of vata.
Medicines which reduces avarodh of rakta and quantitative increase of rakta alongwith which reduces inflammation of siras is helpful in rakta avrita vata.
Raktamokshana reduces quantitative increase of blood. Virechan is the best shodhan procedure for raktadusti. Fluid loss due to virechan also has impact on intravascular quantity of fluid plasma does reducing the avarodh.
Shita pradeha mainly medicines like dashanga lepa, kamala, ushira, yastimadhu, sariva, chandan, padmak, darbha etc drugs helps to reduce local inflammatory response. Anti thrombotic effect of darbha, kamala also reduces the avarodh to the gati of vata.
Medicines which reduces avarodh of rakta and quantitative increase of rakta alongwith which reduces inflammation of siras is helpful in rakta avrita vata.
[06/04 8:54 AM] Katoch sir:
Concept needs clarity Dr Ojha. In raktavrit vaat increase of rakta means whole blood or RBCs ? In phlebitis which Rakta (whole blood or RBCs) is involed with increased volume ?
[06/04 10:03 AM] satyendra ojha sir:
Dr Katoch sir , since Raktaavrita vaata is initial pathogenesis , so it will be specific to particular disease . e.g. in polycythaemia rubra Vera , RBCs with or without WBCs & Platelets are involved. In Apparent Erythrocytosis , only RBC.. In hyperviscosity syndrome , whole blood.. In erythromelalgia , blood vessels . in phlebitis , veins.. In thromboembolism , Platelets and certain clotting factors. Thrombophlebitis , veins with Platelets & certain clotting factors.. Concept is very clear , need to evaluate specifically..
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MD PhD (Kayachikitsa)
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